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dc.contributor.authorLo, HJen_US
dc.contributor.authorWang, JSen_US
dc.contributor.authorLin, CYen_US
dc.contributor.authorChen, CGen_US
dc.contributor.authorHsiao, TYen_US
dc.contributor.authorHsu, CTen_US
dc.contributor.authorSu, CLen_US
dc.contributor.authorFann, MJen_US
dc.contributor.authorChing, YTen_US
dc.contributor.authorYang, YLen_US
dc.date.accessioned2014-12-08T15:19:34Z-
dc.date.available2014-12-08T15:19:34Z-
dc.date.issued2005-03-01en_US
dc.identifier.issn0066-4804en_US
dc.identifier.urihttp://dx.doi.org/10.1128/AAC.49.3.1213-1215.2005en_US
dc.identifier.urihttp://hdl.handle.net/11536/13933-
dc.description.abstractThe ERG3 gene in Candida albicans was identified as a gene whose mRNA level was higher in the cph1/cph1 efg1/efg1 double mutant than in the wild-type cells. Further study showed that Efg1, but not Cph1, negatively regulated ERG3. Mutations in EFG1 consistently increased the susceptibility of the cells to antifungal agents.en_US
dc.language.isoen_USen_US
dc.titleEfg1 involved in drug resistance by regulating the expression of ERG3 in Candida albicansen_US
dc.typeArticleen_US
dc.identifier.doi10.1128/AAC.49.3.1213-1215.2005en_US
dc.identifier.journalANTIMICROBIAL AGENTS AND CHEMOTHERAPYen_US
dc.citation.volume49en_US
dc.citation.issue3en_US
dc.citation.spage1213en_US
dc.citation.epage1215en_US
dc.contributor.department生物科技學系zh_TW
dc.contributor.department資訊工程學系zh_TW
dc.contributor.departmentDepartment of Biological Science and Technologyen_US
dc.contributor.departmentDepartment of Computer Scienceen_US
dc.identifier.wosnumberWOS:000227339500054-
dc.citation.woscount10-
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